Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system

R. L. Schnaar; A. Yamamoto; K. Furukawa; S. Yamashiro; M. Shin; M. Okada; S. Fukumoto; M. Haraguchi; N. Takeda; K. Fujimura; M. Sakae; M. Kishikawa; H. Shiku; K. Furukawa; S. Aizawa

Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system

R. L. Schnaar, A. Yamamoto, K. Furukawa, S. Yamashiro, M. Shin, M. Okada, S. Fukumoto, M. Haraguchi, N. Takeda, K. Fujimura, M. Sakae, M. Kishikawa, H. Shiku, K. Furukawa, S. Aizawa

研究成果: ジャーナルへの寄稿 › 総説 › 査読

抄録

Mice genetically engineered to lack complex brain gangliosides developed normal brain anatomy, histology, and ultrastructure, although they demonstrated a modest change in central nervous system conduction velocity.

本文言語	英語
ページ（範囲）	195-200
ページ数	6
ジャーナル	Chemtracts
巻	11
号	3
出版ステータス	出版済み - 3月 1998
外部発表	はい

!!!All Science Journal Classification (ASJC) codes

化学一般
生化学
分子生物学

他のファイルとリンク

引用スタイル

@article{79e97ab7274045c38d974fa77c08a6c2,

title = "Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system",

abstract = "Mice genetically engineered to lack complex brain gangliosides developed normal brain anatomy, histology, and ultrastructure, although they demonstrated a modest change in central nervous system conduction velocity.",

author = "Schnaar, {R. L.} and A. Yamamoto and K. Furukawa and S. Yamashiro and M. Shin and M. Okada and S. Fukumoto and M. Haraguchi and N. Takeda and K. Fujimura and M. Sakae and M. Kishikawa and H. Shiku and K. Furukawa and S. Aizawa",

year = "1998",

month = mar,

language = "English",

volume = "11",

pages = "195--200",

journal = "Chemtracts",

issn = "1431-9268",

publisher = "Data Trace Publishing Co.",

number = "3",

}

TY - JOUR

T1 - Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system

AU - Schnaar, R. L.

AU - Yamamoto, A.

AU - Furukawa, K.

AU - Yamashiro, S.

AU - Shin, M.

AU - Okada, M.

AU - Fukumoto, S.

AU - Haraguchi, M.

AU - Takeda, N.

AU - Fujimura, K.

AU - Sakae, M.

AU - Kishikawa, M.

AU - Shiku, H.

AU - Furukawa, K.

AU - Aizawa, S.

PY - 1998/3

Y1 - 1998/3

N2 - Mice genetically engineered to lack complex brain gangliosides developed normal brain anatomy, histology, and ultrastructure, although they demonstrated a modest change in central nervous system conduction velocity.

AB - Mice genetically engineered to lack complex brain gangliosides developed normal brain anatomy, histology, and ultrastructure, although they demonstrated a modest change in central nervous system conduction velocity.

UR - http://www.scopus.com/inward/record.url?scp=0031768116&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=0031768116&partnerID=8YFLogxK

M3 - Review article

AN - SCOPUS:0031768116

SN - 1431-9268

VL - 11

SP - 195

EP - 200

JO - Chemtracts

JF - Chemtracts

IS - 3

ER -

Mice with disrupted GM2/GD2 synthase gene lack complex gangliosides but exhibit only subtle defects in their nervous system

抄録

!!!All Science Journal Classification (ASJC) codes

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