TY - JOUR
T1 - Hsc70 contributes to cancer cell survival by preventing Rab1A degradation under stress conditions
AU - Tanaka, Masako
AU - Mun, Saya
AU - Harada, Akihito
AU - Ohkawa, Yasuyuki
AU - Inagaki, Azusa
AU - Sano, Soichi
AU - Takahashi, Katsuyuki
AU - Izumi, Yasukatsu
AU - Osada-Oka, Mayuko
AU - Wanibuchi, Hideki
AU - Yamagata, Masayo
AU - Yukimura, Tokihito
AU - Miura, Katsuyuki
AU - Shiota, Masayuki
AU - Iwao, Hiroshi
N1 - Funding Information:
The authors thank Mayomi Okada, Rika Mishima, and Mami Shimojyo (Laboratory of Clinical Pharmacology, Faculty of Pharmacy, Osaka Ohtani University) for their technical support. The authors also wish to thank members of the Central Laboratory of Osaka City University Graduate School of Medicine, for providing technical support. This work was partly performed in the Cooperative Research Project Program of the Medical Institute of Bioregulation, Kyushu University.
PY - 2014/5/6
Y1 - 2014/5/6
N2 - Heat shock cognate protein 70 (Hsc70) acts as a molecular chaperone for the maintenance of intracellular proteins, which allows cancer cells to survive under proteotoxic stress. We attempted to use Hsc70 to identify key molecules in cancer cell survival. Here, we performed mass-spectrometry-based proteomics analysis utilizing affinity purification with anti-Hsc70 antibodies; as a result, 83 differentially expressed proteins were identified under stress conditions. This result implies that there was a change in the proteins with which Hsc70 interacted in response to stress. Among the proteins identified under both serum-depleted and 5-fluorouracil-treated conditions, Rab1A was identified as an essential molecule for cancer cell survival. Hsc70 interacted with Rab1A in a chaperone-dependent manner. In addition, Hsc70 knockdown decreased the level of Rab1A and increased the level of its ubiquitination under stress conditions, suggesting that Hsc70 prevented the degradation of Rab1A denatured by stress exposure. We also found that Rab1A knockdown induced cell death by inhibition of autophagosome formation. Rab1A may therefore contribute to overcoming proteotoxic insults, which allows cancer cells to survive under stress conditions. Analysis of Hsc70 interactors provided insight into changes of intracellular status. We expect further study of the Hsc70 interactome to provide a more comprehensive understanding of cancer cell physiology.
AB - Heat shock cognate protein 70 (Hsc70) acts as a molecular chaperone for the maintenance of intracellular proteins, which allows cancer cells to survive under proteotoxic stress. We attempted to use Hsc70 to identify key molecules in cancer cell survival. Here, we performed mass-spectrometry-based proteomics analysis utilizing affinity purification with anti-Hsc70 antibodies; as a result, 83 differentially expressed proteins were identified under stress conditions. This result implies that there was a change in the proteins with which Hsc70 interacted in response to stress. Among the proteins identified under both serum-depleted and 5-fluorouracil-treated conditions, Rab1A was identified as an essential molecule for cancer cell survival. Hsc70 interacted with Rab1A in a chaperone-dependent manner. In addition, Hsc70 knockdown decreased the level of Rab1A and increased the level of its ubiquitination under stress conditions, suggesting that Hsc70 prevented the degradation of Rab1A denatured by stress exposure. We also found that Rab1A knockdown induced cell death by inhibition of autophagosome formation. Rab1A may therefore contribute to overcoming proteotoxic insults, which allows cancer cells to survive under stress conditions. Analysis of Hsc70 interactors provided insight into changes of intracellular status. We expect further study of the Hsc70 interactome to provide a more comprehensive understanding of cancer cell physiology.
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U2 - 10.1371/journal.pone.0096785
DO - 10.1371/journal.pone.0096785
M3 - Article
C2 - 24801886
AN - SCOPUS:84900412166
SN - 1932-6203
VL - 9
JO - PloS one
JF - PloS one
IS - 5
M1 - e96785
ER -