Focal cortical dysplasia with calcification: A case report

Kazuhiro Samura; Takato Morioka; Fumiaki Yoshida; Kimiaki Hashiguchi; Yasushi Miyagi; Masahiro Mizoguchi; Tadahisa Shono; Shinji Nagata; Satoshi O. Suzuki; Tomio Sasaki

doi:10.1007/s00381-007-0566-4

Focal cortical dysplasia with calcification: A case report

Kazuhiro Samura, Takato Morioka, Fumiaki Yoshida, Kimiaki Hashiguchi, Yasushi Miyagi, Masahiro Mizoguchi, Tadahisa Shono, Shinji Nagata, Satoshi O. Suzuki, Tomio Sasaki

Pathobiology

Research output: Contribution to journal › Article › peer-review

5 Citations (Scopus)

Abstract

Case report: Focal cortical dysplasia (FCD) with calcification is rare. We presented a 13-year-old epileptic patient with FCD and calcification in the left frontal lobe. At age 24, the FCD lesion and the surrounding epileptogenic cortex and underlying subcortex were removed after chronic subdural electrode recording. Histological examination showed that the calcified lesion was not independent of the FCD lesion but located in the subcortical area of the FCD lesion. A neoplastic nature was ruled out for the lesion. Discussion: The pathophysiological mechanism involved in the coexistence of FCD and calcification is discussed.

Original language	English
Pages (from-to)	619-622
Number of pages	4
Journal	Child's Nervous System
Volume	24
Issue number	5
DOIs	https://doi.org/10.1007/s00381-007-0566-4
Publication status	Published - May 2008

All Science Journal Classification (ASJC) codes

Pediatrics, Perinatology, and Child Health
Clinical Neurology

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10.1007/s00381-007-0566-4

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title = "Focal cortical dysplasia with calcification: A case report",

abstract = "Case report: Focal cortical dysplasia (FCD) with calcification is rare. We presented a 13-year-old epileptic patient with FCD and calcification in the left frontal lobe. At age 24, the FCD lesion and the surrounding epileptogenic cortex and underlying subcortex were removed after chronic subdural electrode recording. Histological examination showed that the calcified lesion was not independent of the FCD lesion but located in the subcortical area of the FCD lesion. A neoplastic nature was ruled out for the lesion. Discussion: The pathophysiological mechanism involved in the coexistence of FCD and calcification is discussed.",

author = "Kazuhiro Samura and Takato Morioka and Fumiaki Yoshida and Kimiaki Hashiguchi and Yasushi Miyagi and Masahiro Mizoguchi and Tadahisa Shono and Shinji Nagata and Suzuki, {Satoshi O.} and Tomio Sasaki",

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T2 - A case report

AU - Samura, Kazuhiro

AU - Morioka, Takato

AU - Yoshida, Fumiaki

AU - Hashiguchi, Kimiaki

AU - Miyagi, Yasushi

AU - Mizoguchi, Masahiro

AU - Shono, Tadahisa

AU - Nagata, Shinji

AU - Suzuki, Satoshi O.

AU - Sasaki, Tomio

PY - 2008/5

Y1 - 2008/5

N2 - Case report: Focal cortical dysplasia (FCD) with calcification is rare. We presented a 13-year-old epileptic patient with FCD and calcification in the left frontal lobe. At age 24, the FCD lesion and the surrounding epileptogenic cortex and underlying subcortex were removed after chronic subdural electrode recording. Histological examination showed that the calcified lesion was not independent of the FCD lesion but located in the subcortical area of the FCD lesion. A neoplastic nature was ruled out for the lesion. Discussion: The pathophysiological mechanism involved in the coexistence of FCD and calcification is discussed.

AB - Case report: Focal cortical dysplasia (FCD) with calcification is rare. We presented a 13-year-old epileptic patient with FCD and calcification in the left frontal lobe. At age 24, the FCD lesion and the surrounding epileptogenic cortex and underlying subcortex were removed after chronic subdural electrode recording. Histological examination showed that the calcified lesion was not independent of the FCD lesion but located in the subcortical area of the FCD lesion. A neoplastic nature was ruled out for the lesion. Discussion: The pathophysiological mechanism involved in the coexistence of FCD and calcification is discussed.

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Focal cortical dysplasia with calcification: A case report

Abstract

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