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Dive into the research topics where Shojiro Haji is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Collaborations and top research areas from the last five years
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Role of a transmembrane protein, epithelial membrane protein 1, in the pathogenesis of pancreatic ductal adenocarcinoma
Ohno, A., Fujimori, N., Matsumoto, K., Haji, S., Sugahara, O., Takeno, A., Yamamoto, T., Suenaga, A., Kakehashi, S., Ueda, T., Murakami, M., Teramatsu, K., Ueda, K., Oono, T., Oda, Y., Nakayama, K. I. & Ogawa, Y., Jan 29 2026, In: Oncogene. 45, 2, p. 307-321 15 p.Research output: Contribution to journal › Article › peer-review
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Condensation-dependent interactome of a chromatin remodeler underlies tumor suppressor activities
Tsukamoto, Y., Kawamura, A., Yurtsever, A., Suzuki, H., Rojas-Chaverra, N. M., Sato, H., Ino, D., Ichikawa, T., Wei, W., Haji, S., Voon, D. C. C., Matsumoto, A., Matsumoto, K., Takahashi, H., Kodera, N., Fukuma, T., Ogawa, Y., Nishiyama, M. & Sakai, K., Dec 2025, In: Nature communications. 16, 1, 9599.Research output: Contribution to journal › Article › peer-review
Open Access -
Efficacy and Safety of Rituximab plus Modified EPOCH (Etoposide, Vincristine, Doxorubicin, Carboplatin, and Prednisolone) for Transplant-ineligible Relapsed/Refractory Diffuse Large B-cell Lymphoma
Fujioka, E., Kiyasu, J., Choi, I., Yagi, Y., Sawabe, T., Oyama, M., Hoashi, K., Tsuda, M., Takamatsu, A., Haji, S., Yufu, Y., Suehiro, Y. & Shiratsuchi, M., Oct 15 2025, In: Internal Medicine. 64, 20, p. 2953-2959 7 p.Research output: Contribution to journal › Article › peer-review
Open Access -
Multiomics analysis unveils the cellular ecosystem with clinical relevance in aldosterone-producing adenomas with KCNJ5 mutations
Yokomoto-Umakoshi, M., Fujita, M., Umakoshi, H., Ogasawara, T., Iwahashi, N., Nakatani, K., Kaneko, H., Fukumoto, T., Nakao, H., Haji, S., Kawamura, N., Shimma, S., Seki, M., Suzuki, Y., Izumi, Y., Oda, Y., Eto, M., Ogawa, S., Bamba, T. & Ogawa, Y., Mar 4 2025, In: Proceedings of the National Academy of Sciences of the United States of America. 122, 9, e2421489122.Research output: Contribution to journal › Article › peer-review
Open Access9 Link opens in a new tab Citations (Scopus) -
TET3-overexpressing macrophages are a unifying pathogenic feature with therapeutic potential in chronic inflammatory diseases
Haji, S. & Ogawa, Y., Nov 3 2025, In: Journal of Clinical Investigation. 135, 21, e198802.Research output: Contribution to journal › Article › peer-review
Open Access